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Aβ is formed from the cleavage of APP (Amyloid Precursor Protein), a large transmembrane protein. Cleavage of APP by β- and then γ-secretases results in the formation of Aβ. Aβ can aggregate to produce amyloid-β oligomers, which are thought to be highly neurotoxic. Over time Aβ can further aggregate to produce the characteristic senile plaques present in Alzheimer's Disease. Aβ can be degraded by enzymes such as neprilysin, insulin degrading enzyme or endothelin converting enzyme. At physiological levels Aβ may be involved in controlling synaptic activity and neuronal survival.
Beta Amyloid and related peptides
Beta Amyloid (1-42) Human 0.1mg
Beta Amyloid (1-42) Human 0.5mg
Beta Amyloid (1-42) Human 1mg
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